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Traditional Medications & Treatments for Alpha1 Antitrypsin Deficiency

Comprehensive Guide to Current and Emerging AATD Therapies


Introduction

Treatment for Alpha-1 Antitrypsin Deficiency has evolved significantly—and continues to advance rapidly.

From traditional augmentation therapy to emerging gene therapies and novel approaches, this guide covers everything you need to know about current treatments, what's on the horizon, and how to make informed decisions about your care.

Understanding your treatment options empowers you to work with your healthcare team to create the best plan for your unique situation.


Traditional Augmentation Therapy

What is AAT Augmentation Therapy?

Alpha-1 Antitrypsin (AAT) augmentation therapy is the primary, FDA-approved treatment specifically for AATD. It involves regular infusions of purified human AAT protein to raise the level of circulating Alpha-1 Antitrypsin in the body, restoring the protease/antiprotease balance.

Think of it as:

  • Replacing what your body can't produce enough of on its own
  • Similar to insulin for diabetes—providing what's missing
  • Protective therapy preventing tissue damage from uncontrolled proteases
  • Long-term disease modification, not just symptom relief

FDA-Approved AAT Products

Currently, there are four FDA-approved medications for AAT augmentation therapy:

1. Prolastin-C (Alpha-1 Proteinase Inhibitor [Human])
  • Manufacturer: Grifols
  • Formulation: Lyophilized (freeze-dried) powder for reconstitution
  • Dosing: 60 mg/kg body weight once weekly
  • Infusion Time: Approximately 15-30 minutes
  • Storage: Refrigerated or room temperature
  • Note: Most widely prescribed AAT therapy globally
2. Aralast NP (Alpha-1 Proteinase Inhibitor [Human])
  • Manufacturer: Takeda (formerly Baxalta/Baxter)
  • Formulation: Lyophilized powder for reconstitution
  • Dosing: 60 mg/kg body weight once weekly
  • Infusion Time: Approximately 15 minutes
  • Storage: Room temperature (up to 25°C)
  • Note: NP = "new process" with enhanced viral inactivation
3. Zemaira (Alpha-1 Proteinase Inhibitor [Human])
  • Manufacturer: CSL Behring
  • Formulation: Liquid solution (ready to use, no reconstitution)
  • Dosing: 60 mg/kg body weight once weekly
  • Infusion Time: Approximately 15 minutes or by rapid push (under 10 minutes)
  • Storage: Refrigerated
  • Note: Convenience of liquid formulation, no mixing required
4. Glassia (Alpha-1 Proteinase Inhibitor [Human])
  • Manufacturer: Takeda
  • Formulation: Liquid solution
  • Dosing: 60 mg/kg body weight once weekly
  • Infusion Time: Approximately 10-20 minutes
  • Storage: Refrigerated
  • Note: Designed for rapid infusion or home administration

How AAT Augmentation Works

The Science:

  1. Source: All FDA-approved products are derived from pooled human plasma from thousands of donors
  2. Purification: Extensive processing removes viruses, bacteria, and other proteins, leaving purified AAT
  3. Administration: Infused into bloodstream (IV infusion)
  4. Distribution: AAT circulates in blood and reaches lung tissue (primary target)
  5. Function: Inhibits neutrophil elastase and other proteases that damage tissue
  6. Duration: Levels remain protective for approximately one week
  7. Repeat: Weekly infusions maintain consistent protective levels

What It Does:

  • Slows lung function decline
  • Reduces frequency of COPD exacerbations
  • Decreases lung inflammation
  • May reduce mortality risk
  • Potentially protects other organ systems
  • May prevent disease progression if started early

What It Doesn't Do:

  • Doesn't reverse existing lung damage
  • Doesn't cure AATD (genetic condition remains)
  • Doesn't eliminate need for other treatments (bronchodilators, etc.)
  • Doesn't guarantee prevention of all complications

Who Should Receive Augmentation Therapy?

Current FDA Approval / Traditional Guidelines:

Approved For:

  • Adults with AATD (specifically ZZ, SZ, or other rare phenotypes with very low AAT levels)
  • Evidence of emphysema on imaging or pulmonary function testing
  • Typically FEV1 between 35-60% predicted (though guidelines vary)
  • Non-smokers or former smokers

Traditional Exclusion Criteria:

  • Active smokers (must quit before therapy)
  • No lung disease present
  • Normal or near-normal lung function
  • Liver disease as only manifestation
  • MZ carriers (usually)
Evolving Perspective / Mark Egly Foundation Advocacy:

Arguments for Earlier/Broader Treatment:

Prevention vs. Waiting for Damage:

  • Why wait for FEV1 to drop to 60% when tissue damage is preventable?
  • Evidence suggests earlier treatment preserves function better
  • Once lung tissue is destroyed, it can't be regenerated
  • Prevention aligns with Hippocratic principle: "prevent disease whenever possible"

Systemic Benefits Beyond Lungs:

  • AATD affects multiple organs, not just lungs
  • AAT has anti-inflammatory effects systemically
  • May prevent autoimmune disease development
  • Potential cancer prevention through immune optimization
  • Neurological protection (Alzheimer's prevention)
  • Liver protection even without overt liver disease

Mark's Vision:
"It is Mark and the Foundation's dream that Alpha1 Antitrypsin becomes the equivalent of the safest and healthiest 'vaccine type' treatment many individuals could receive throughout their entire lifetime."

Current Reality:

  • Insurance typically requires significant lung damage before approval
  • Standards of care are changing slowly
  • Advocacy efforts underway to expand treatment criteria
  • Some physicians prescribe "off-label" for preventive purposes

Administration Methods

Infusion Center / Clinic Administration

Pros:

  • Medical supervision during infusion
  • Immediate help if adverse reaction
  • No home setup required
  • Social aspect (meet other patients)
  • Vitals monitored

Cons:

  • Travel to infusion center weekly
  • Time commitment (infusion + travel)
  • Less flexibility in scheduling
  • Potential exposure to illnesses in clinic

Typical Process:

  1. Arrive at infusion center
  2. Vitals checked (blood pressure, pulse, temp, O2)
  3. IV line placed
  4. Infusion started (15-30 minutes)
  5. Monitored during and after infusion
  6. IV removed, vitals rechecked
  7. Home (total time: 1-2 hours typically)
Home Infusion

Pros:

  • Convenience of home treatment
  • Flexible scheduling
  • No travel required
  • Privacy and comfort
  • Reduced infection exposure
  • Time savings

Cons:

  • Requires training and confidence
  • Home storage of medication
  • Self-management of equipment
  • Need emergency plan if complications
  • May feel isolated (vs. clinic social aspect)

Requirements:

  • Physician approval
  • Completion of training program
  • Home health nurse visits initially
  • Appropriate storage space (refrigerator)
  • Emergency contact plan

Process:

  • Medication delivered to home monthly
  • Patient self-administers or has caregiver assist
  • IV access (peripheral IV, port, or PICC line)
  • Self-monitoring
  • Coordination with home health agency
Subcutaneous Administration (Investigational)

Currently in Clinical Trials:

  • Weekly or more frequent self-injections under skin (like insulin)
  • Potentially higher dosing frequency with smaller volumes
  • Early studies show promise
  • May improve convenience and patient preference
  • Not yet FDA approved

Side Effects and Management

Common Side Effects (Generally Mild):

During/Immediately After Infusion:

  • Headache (most common)
  • Dizziness or lightheadedness
  • Nausea
  • Fatigue
  • Chills or feeling cold
  • Flushing or warmth
  • Mild fever
  • Muscle aches

Management:

  • Slow infusion rate
  • Pre-medication (acetaminophen, antihistamine)
  • Hydration before/after
  • Usually resolve within hours

Injection Site Issues:

  • Bruising where IV placed
  • Tenderness or soreness
  • Mild swelling

Management:

  • Rotate IV sites
  • Apply warm compress
  • Usually minor and temporary
Rare but Serious Side Effects:

Allergic Reactions:

  • Hives, itching, rash
  • Difficulty breathing, wheezing
  • Chest tightness
  • Swelling of face, lips, tongue
  • Severe hypotension

Action: Stop infusion immediately, seek emergency care

Viral Transmission Risk:

  • Extremely rare with modern purification
  • All products undergo extensive viral inactivation
  • Theoretical risk from plasma-derived product
  • No documented HIV or hepatitis transmission with current products

IgA Deficiency Concern:

  • Patients with IgA deficiency may have antibodies against IgA
  • Risk of anaphylactic reaction
  • Should be tested before starting therapy
Long-Term Considerations:

Ongoing Monitoring:

  • Regular pulmonary function tests
  • AAT trough levels (measure before next infusion)
  • Liver function tests
  • Monitoring for adverse effects

Vein Health:

  • Repeated IV access can damage veins over time
  • May need port or PICC line eventually
  • Rotation of sites important
  • Proper hydration helps vein health

Cost and Insurance Coverage

The Financial Reality:

Annual Cost:

  • Approximately $100,000 - $200,000+ per year
  • Varies by product, dosage (weight-based), and pharmacy
  • One of the most expensive chronic therapies

Why So Expensive:

  • Plasma collection and testing from thousands of donors
  • Extensive purification and viral inactivation processes
  • Limited patient population (rare disease, limited market)
  • Manufacturing complexity
  • Distribution and storage requirements
Insurance Coverage:

Most insurance plans cover AAT therapy when:

  • FDA-approved indication (emphysema with documented lung disease)
  • Pre-authorization obtained
  • Medical necessity documented
  • Smoking cessation proven
  • Proper diagnosis and testing

Challenges:

  • Prior authorization can be lengthy and complex
  • Criteria vary by insurer
  • Denials common initially (appeals often successful)
  • Documentation requirements extensive
  • Some plans have step therapy requirements

Medicare Coverage:

  • Part B covers IV infusions in clinic/hospital
  • Part D may cover home infusions
  • Supplemental coverage often needed for copays
Financial Assistance Programs:

Pharmaceutical Company Programs:

  • CSL Behring Support: For Zemaira patients - copay assistance, reimbursement support
  • Grifols Plasma Alliance: For Prolastin-C - patient assistance
  • Takeda Patient Support: For Aralast NP and Glassia - copay cards, financial assistance
  • Octapharma: Various support programs

General Assistance:

  • HealthWell Foundation - Copay assistance for AATD
  • Patient Advocate Foundation - Insurance navigation and copay relief
  • AlphaNet - Case management, insurance coordination
  • Alpha-1 Foundation - Resources and referrals
  • Mark Egly Foundation - Resource connections

Getting Help:

  • Contact AlphaNet coordinator (1-800-577-2586) - FREE service helping with insurance
  • Specialty pharmacies often have financial coordinators
  • Patient advocate services
  • Hospital financial counselors

Supportive Lung Treatments

Bronchodilators

Purpose: Open airways, reduce breathlessness, improve lung function

Types:

Short-Acting Beta-Agonists (SABAs):

  • Albuterol (ProAir, Ventolin, Proventil)
  • Levalbuterol (Xopenex)
  • Use: Quick relief for acute symptoms ("rescue inhaler")
  • Duration: 4-6 hours

Long-Acting Beta-Agonists (LABAs):

  • Salmeterol (Serevent)
  • Formoterol (Foradil, Perforomist)
  • Use: Maintenance, twice daily
  • Duration: 12 hours
  • Note: Should not be used alone (combine with corticosteroid)

Short-Acting Anticholinergics:

  • Ipratropium (Atrovent)
  • Use: Quick relief, maintenance
  • Duration: 4-6 hours

Long-Acting Anticholinergics (LAMAs):

  • Tiotropium (Spiriva)
  • Aclidinium (Tudorza)
  • Umeclidinium (Incruse)
  • Glycopyrrolate (Seebri)
  • Use: Once-daily maintenance
  • Duration: 24 hours

Combination Inhalers:

  • LABA + Corticosteroid: Advair, Symbicort, Breo, others
  • LABA + LAMA: Anoro, Stiolto, Bevespi
  • LABA + LAMA + Corticosteroid: Trelegy
  • Advantage: Convenience, better adherence, synergistic effects

Inhaled Corticosteroids (ICS)

Purpose: Reduce airway inflammation

Common Options:

  • Fluticasone (Flovent)
  • Budesonide (Pulmicort)
  • Mometasone (Asmanex)
  • Ciclesonide (Alvesco)

Use: Maintenance therapy, typically twice daily

Note: Controversial in AATD—some evidence suggests less benefit than in other COPD, but individual response varies

Oral Corticosteroids

Purpose: Reduce inflammation during exacerbations

Common: Prednisone, methylprednisolone

Use: Short courses (5-10 days) for acute worsening, NOT long-term due to side effects

Phosphodiesterase-4 (PDE4) Inhibitor

Roflumilast (Daliresp)

  • Reduces inflammation
  • For severe COPD with chronic bronchitis and frequent exacerbations
  • Oral medication, once daily
  • Can have GI side effects

Mucolytics

Purpose: Thin mucus, make it easier to cough up

Guaifenesin (Mucinex):

  • Over-the-counter
  • Helps loosen chest congestion
  • Adequate hydration important

N-Acetylcysteine (NAC):

  • Antioxidant properties
  • Thins mucus
  • Some evidence for reducing exacerbations
  • Available as supplement or prescription

Antibiotics

Purpose: Treat bacterial lung infections

When Used:

  • Acute exacerbations with signs of infection
  • Chronic lung infections (bronchiectasis)
  • Prophylactic use in some cases of frequent infections

Common Options:

  • Azithromycin (often used long-term for anti-inflammatory effects)
  • Doxycycline
  • Amoxicillin/clavulanate
  • Fluoroquinolones (for serious infections)

Oxygen Therapy

Purpose: Maintain adequate blood oxygen levels

Indications:

  • Resting O2 saturation ≤88%
  • Exertional desaturation
  • Sleep-related hypoxemia
  • Improved survival with long-term oxygen when indicated

Delivery Systems:

  • Compressed oxygen tanks (portable and stationary)
  • Liquid oxygen systems
  • Oxygen concentrators (home use)
  • Portable oxygen concentrators (POCs) - battery powered, travel-friendly

Duration:

  • Continuous (24/7)
  • Only with exertion
  • Only during sleep
  • As prescribed based on individual needs

Liver-Specific Treatments

For AATD-Related Liver Disease

No Specific Medication for AATD Liver Disease:

  • AAT augmentation does NOT treat liver manifestations
  • Management is supportive and preventive

Strategies:

Lifestyle Modifications:

  • Avoid alcohol completely
  • Maintain healthy weight
  • Avoid hepatotoxic medications when possible
  • Vaccination (hepatitis A & B)
  • Monitor liver function regularly

Treating Complications:

  • Portal hypertension management
  • Varices screening and treatment
  • Ascites management (diuretics, paracentesis)
  • Hepatic encephalopathy treatment (lactulose, rifaximin)
  • Nutritional support

Liver Transplantation:

  • Definitive treatment for end-stage AATD liver disease
  • Excellent outcomes (80-90% 5-year survival)
  • Corrects genetic defect in liver (but recipient still makes abnormal AAT from transplanted liver)
  • Lungs not affected by liver transplant

Autoimmune & Inflammatory Treatments

For AATD-Related Autoimmune Conditions

Approach Depends on Specific Condition:

Rheumatoid Arthritis, Lupus, Other Autoimmune Diseases:

  • Disease-modifying antirheumatic drugs (DMARDs)
  • Biologic agents (TNF inhibitors, etc.)
  • Immunosuppressants
  • Corticosteroids (short-term)

AATD Consideration:

  • AAT augmentation may help autoimmune symptoms (under investigation)
  • Standard autoimmune treatments used
  • Immunosuppression may increase infection risk (monitor closely with lung disease)

Anti-Inflammatory Strategies:

  • Diet (Mediterranean, anti-inflammatory focus)
  • Omega-3 fatty acids
  • Exercise (appropriate level)
  • Stress management
  • Smoking cessation
  • Limiting alcohol

Pulmonary Rehabilitation

Comprehensive Exercise and Education Program

What It Includes:

  • Supervised exercise training
  • Breathing techniques
  • Nutritional counseling
  • Energy conservation strategies
  • Education about lung disease
  • Psychosocial support
  • Goal setting and self-management

Benefits:

  • Improved exercise capacity
  • Reduced breathlessness
  • Better quality of life
  • Fewer hospitalizations
  • Increased confidence and independence
  • Enhanced ability to perform daily activities

Duration: Typically 6-12 weeks, 2-3 sessions per week

Location: Hospital-based, outpatient centers, some home programs

Insurance: Usually covered with referral


Lung Volume Reduction

Surgical and Non-Surgical Options

For Severe Emphysema in Select Patients:

Surgical Lung Volume Reduction (LVRS)
  • Removal of most damaged lung tissue
  • Allows remaining healthier tissue to function better
  • Major surgery with risks
  • Benefit in carefully selected patients (particularly upper lobe predominant disease)
Bronchoscopic Lung Volume Reduction (BLVR)
  • Endobronchial Valves: One-way valves placed in airways to collapse diseased areas
  • Less invasive than surgery
  • For heterogeneous emphysema
  • Growing body of evidence for efficacy
Bullectomy
  • Surgical removal of large bullae (air pockets)
  • Can improve lung function if bullae compressing healthy tissue

Lung Transplantation

Definitive Treatment for End-Stage Lung Disease

When Considered:

  • FEV1 <25% predicted
  • Rapid decline despite maximal therapy
  • Frequent hospitalizations
  • Hypoxemia or hypercapnia
  • Severely limited quality of life
  • No other treatment options

Process:

  • Referral to transplant center
  • Extensive evaluation (medical, psychological, social)
  • Listing with UNOS (United Network for Organ Sharing)
  • Wait time varies (months to years)
  • Lung allocation score determines priority

Types:

  • Single lung transplant
  • Double lung transplant (more common in AATD)

Outcomes:

  • Good short-term survival (80-90% at 1 year)
  • Median survival 5-6 years
  • Some patients live 10, 15, 20+ years post-transplant
  • Significantly improved quality of life
  • Requires lifelong immunosuppression

Considerations:

  • Major surgery with significant risks
  • Lifelong anti-rejection medications
  • Risk of infections, rejection, complications
  • Extensive medical follow-up required
  • Lifestyle changes necessary
  • Genetic AATD not cured (but new lungs make normal AAT)

Emerging and Future Therapies

What's on the Horizon

Gene Therapy

Goal: Correct the genetic defect causing AATD

Approaches:

  • Gene Addition: Adding functional SERPINA1 gene
  • Gene Editing: CRISPR or other technologies to fix mutation
  • Delivery: Viral vectors, nanoparticles, direct injection

Status: Multiple clinical trials ongoing

  • Some showing promise in early phases
  • Years away from FDA approval
  • Could potentially cure AATD with one-time treatment

Potential Benefits:

  • Eliminate need for weekly infusions
  • Correct underlying genetic cause
  • Produce AAT naturally in body
  • Prevent all organ manifestations

Challenges:

  • Delivery to enough cells to make sufficient AAT
  • Long-term expression and safety
  • Immune response to vectors
  • Cost and accessibility
Alternative AAT Production Methods

Recombinant AAT:

  • Produced in cell cultures or transgenic animals rather than human plasma
  • Unlimited supply potential
  • No viral transmission risk
  • Lower cost possible
  • In development, not yet approved

Plant-Based Production:

  • AAT produced in tobacco, corn, or other plants
  • Scalable production
  • Very low cost potential
  • Early research phase

Transgenic Animal Production:

  • Goats, cows producing human AAT in milk
  • Proven feasible in research
  • Regulatory pathway unclear
Inhaled AAT

Rationale: Deliver AAT directly to lungs where needed most

Status: Clinical trials with mixed results

  • Some formulations showing promise
  • May complement IV therapy
  • Could be preventive in early disease

Advantages:

  • Direct delivery to target organ
  • Potentially more convenient
  • Lower systemic dose needed
Oral AAT

Challenge: Protein degraded by digestive system

Approaches Being Investigated:

  • Protected formulations
  • Absorption enhancers
  • Very early research
Small Molecule Drugs

Goal: Drugs that enhance AAT production or function

Approaches:

  • Compounds that increase AAT gene expression
  • Molecules that improve AAT folding and secretion
  • Protease inhibitors (non-AAT based)

Status: Preclinical and early clinical research

Stem Cell Therapy

Goal: Replace AAT-deficient cells with normal cells

Approaches:

  • Liver stem cells
  • Induced pluripotent stem cells (iPSCs) corrected and reimplanted
  • Very early research
Antisense Oligonucleotides (ASOs)

For Liver Disease Specifically:

  • Reduce production of misfolded Z protein accumulating in liver
  • Proof of concept in trials
  • May prevent/treat liver disease manifestation

Complementary and Integrative Approaches

Evidence-Informed Supportive Therapies

Important Note: These should complement, not replace, standard medical care. Discuss with your physician before starting any complementary therapy.

Nutrition

Anti-Inflammatory Diet:

  • Mediterranean diet pattern
  • Omega-3 rich foods (fatty fish, walnuts, flaxseed)
  • Colorful fruits and vegetables
  • Whole grains
  • Limit processed foods, red meat, sugar

Adequate Protein:

  • Important for muscle maintenance, immune function
  • Lean proteins, plant proteins
  • May need increased intake with lung disease

Antioxidants:

  • Vitamin C, E, beta-carotene from food sources
  • Colorful vegetables and fruits
  • Some evidence for benefit in lung health

Hydration:

  • Essential for mucus clearance
  • 8+ glasses daily unless restricted
  • Warm beverages may be soothing

Supplements

N-Acetylcysteine (NAC):

  • Antioxidant and mucolytic
  • Some evidence for reducing exacerbations
  • Generally safe
  • Typical dose 600mg 1-2x daily

Vitamin D:

  • Many COPD patients deficient
  • Important for bone health, immune function
  • Test levels, supplement if low

Omega-3 Fatty Acids:

  • Anti-inflammatory
  • May reduce systemic inflammation
  • Fish oil or algae-based

Vitamin C and E:

  • Antioxidants
  • May help lung health
  • Food sources preferred, supplements if needed

Coenzyme Q10 (CoQ10):

  • Antioxidant
  • May help with energy/fatigue
  • Limited evidence but generally safe

Probiotics:

  • May support immune function
  • Gut health important for overall wellness

ALWAYS discuss supplements with physician—can interact with medications

Mind-Body Practices

Yoga:

  • Gentle stretching, breathing focus
  • Can improve flexibility, stress, breathing control
  • Choose classes appropriate for limitations

Tai Chi:

  • Gentle movement meditation
  • Evidence for improving balance, strength, breathing in COPD
  • Low impact, very safe

Meditation & Mindfulness:

  • Stress reduction
  • May improve coping, quality of life
  • Pain management
  • Many free apps and resources

Breathing Techniques:

  • Pursed-lip breathing
  • Diaphragmatic breathing
  • Reduces breathlessness, improves efficiency
  • Taught in pulmonary rehabilitation

Acupuncture

  • May help with breathlessness, stress
  • Limited but some positive evidence
  • Generally safe when performed by licensed acupuncturist

Massage Therapy

  • Relaxation, stress relief
  • May help with muscle tension
  • Generally safe
  • Choose therapist familiar with medical conditions

Treatment Decision-Making

Working with Your Healthcare Team

Important Questions to Ask:

About AAT Augmentation Therapy:

  • Am I a candidate for augmentation therapy?
  • What are the potential benefits in my case?
  • What are the risks and side effects?
  • How is it administered—clinic vs. home?
  • How long will I need therapy?
  • How will we monitor effectiveness?
  • What if I can't get insurance approval?

About Other Treatments:

  • Why are you recommending this medication?
  • How does it work?
  • What are alternatives?
  • What side effects should I watch for?
  • How will we know if it's working?
  • Are there interactions with other medications?

General:

  • What's my prognosis with treatment vs. without?
  • When should I consider more aggressive therapies?
  • Are there clinical trials I should consider?
  • What lifestyle changes are most important?
  • How often should I be monitored?

When to Start Treatment

Traditional Approach:

  • Wait for significant lung function decline (FEV1 <60%)
  • Documented emphysema on CT scan
  • Symptoms impacting quality of life

Preventive Approach (Mark Egly Foundation Advocacy):

  • Earlier intervention before damage occurs
  • Consider AAT therapy even with normal lung function
  • Focus on preventing complications, not just treating them
  • Systemic benefits beyond lungs

Factors to Consider:

  • Genotype (ZZ, SZ more aggressive)
  • Age (younger = more years at risk)
  • Family history (severe disease in relatives?)
  • Baseline lung function trend
  • Presence of symptoms
  • Other organ involvement
  • Personal values and preferences
  • Insurance and financial considerations

Treatment Goals

Primary Goals:

  • Slow disease progression
  • Prevent or minimize lung function decline
  • Reduce exacerbations
  • Maintain quality of life
  • Optimize functional capacity
  • Prevent complications
  • Maximize lifespan

Setting Realistic Expectations:

  • Treatment slows decline, doesn't reverse damage
  • Some decline may occur despite best therapy
  • Individual responses vary
  • Requires long-term commitment
  • Combines medical treatment with lifestyle optimization

Living Well with Treatment

Treatment Adherence

Why It Matters:

  • Consistency is key to effectiveness
  • Missing doses reduces protection
  • Lifestyle factors impact outcomes as much as medication

Strategies for Adherence:

  • Integrate treatment into routine
  • Use reminders (phone alarms, apps)
  • Prepare medications weekly
  • Address barriers proactively (cost, side effects, access)
  • Understand WHY you're taking each medication
  • Track adherence to identify patterns
  • Communicate with team about challenges

Monitoring Your Treatment

Regular Assessments:

  • Pulmonary function tests (spirometry) - typically every 3-6 months
  • AAT trough levels - annually or as needed
  • Liver function tests - at least annually
  • CT scans - as indicated
  • Six-minute walk test - tracking exercise capacity
  • Quality of life questionnaires

Self-Monitoring:

  • Daily symptoms - cough, breathlessness, energy
  • Pulse oximetry at home if appropriate
  • Weight tracking
  • Exacerbation frequency
  • Functional abilities
  • Overall well-being

Signs Your Treatment May Need Adjustment:

  • Increasing breathlessness
  • More frequent exacerbations
  • Declining exercise tolerance
  • Declining lung function on testing
  • New symptoms
  • Medication side effects affecting quality of life

Financial Navigation

Making Treatment Affordable

Strategies:

  1. Work with AlphaNet Coordinator (FREE service)
    • Expert insurance navigation
    • Prior authorization assistance
    • Appeals support
    • Resource connections
  2. Apply for Patient Assistance Programs
    • Pharmaceutical company programs
    • HealthWell Foundation
    • Patient Advocate Foundation
  3. Specialty Pharmacy Selection
    • Compare costs and services
    • Many offer financial coordinators
    • Home delivery options
  4. Optimize Insurance
    • Choose plans with best chronic disease coverage
    • Understand deductibles, out-of-pocket maximums
    • Timing of care around calendar year
  5. Tax Deductions
    • Medical expenses >7.5% of AGI are deductible
    • Keep meticulous records
    • Consult tax professional
  6. Clinical Trials
    • Investigational treatments provided free
    • Close monitoring included
    • Contributing to research

Resources for Treatment Information

AATD Organizations:

  • Alpha-1 Foundation: Treatment guidelines, specialist directory
  • AlphaNet: Case management, treatment coordination (FREE)
  • Mark Egly Foundation: Ask the MD, treatment decision support

Pharmacy Patient Support:

  • Each manufacturer has patient support programs
  • Financial assistance applications
  • Copay cards and assistance

Clinical Trials:

  • ClinicalTrials.gov - search "Alpha-1 Antitrypsin"
  • Ask your AATD specialist about current trials
  • Alpha-1 Foundation research updates

The Future is Promising

Treatment for AATD has never been more hopeful:

✅ Four FDA-approved augmentation therapies available now
✅ Gene therapy in clinical trials - potential cure on horizon
✅ Alternative AAT production methods in development
✅ Growing understanding of systemic benefits
✅ Advocacy for earlier treatment gaining traction
✅ Novel delivery methods being investigated
✅ Research into autoimmune and inflammatory connections
✅ Improved lung transplant outcomes

The standard of care is evolving rapidly.

The Mark Egly Foundation is at the forefront of advocating for:

  • Earlier treatment initiation
  • Expansion of treatment criteria beyond traditional lung-only focus
  • Recognition of systemic benefits
  • Preventive rather than reactive approach
  • Access and affordability improvements

Take Control of Your Treatment

You are not a passive recipient of medical care—you are an active partner in your treatment decisions.

Empower yourself:

  • 📚 Educate yourself about options
  • 🤝 Build a strong healthcare team
  • 💬 Ask questions until you understand
  • 🎯 Set clear treatment goals
  • 📊 Monitor your progress
  • 🔄 Advocate for adjustments when needed
  • 🌐 Connect with others on similar treatments
  • 💪 Take charge of lifestyle factors you can control

Your treatment plan should reflect your values, goals, and circumstances—not just a one-size-fits-all protocol.


Contact Us for Support

Questions about treatments? Need help navigating decisions?

Mark Egly Foundation Resources:

  • 🩺 Ask the MD: Expert physician guidance - markeglyfoundation.org/ask-the-md
  • 🤝 Peer Mentors: Connect with patients on similar treatments
  • 📚 Educational Materials: Treatment decision guides
  • 💬 Community Forums: Discuss treatment experiences with peers
  • 📞 Phone: 1-800-MARK-EGLY
  • ✉️ Email: treatments@markeglyfoundation.org

AlphaNet (FREE Case Management):

  • 📞 1-800-577-2586
  • 🌐 alphanet.org

Alpha-1 Foundation:

  • 📞 1-800-GO-ALPHA-1
  • 🌐 alpha1.org

Traditional treatments offer protection. Emerging therapies offer hope. Your active participation offers the best outcomes.

Together, we're not just treating AATD—we're transforming what's possible for everyone affected by Alpha-1 Antitrypsin Deficiency.


Last Updated: December 2025
This information is for educational purposes and should not replace consultation with your healthcare providers.